Their More moderen work further disclosed that the atypical GTPase domain of Fbx4 could serve as a substrate-binding motif with the SCF E3 ligase intricate and bind to a globular area of TRF1 for its ubiquitination and degradation. These conclusions deliver clues towards the underlying system for managing TRF1, whose https://marcouvwvu.blog2news.com/28841569/a-secret-weapon-for-gençlik-aşısı-nedir